Impairment of IGF-I Expression and Anabolic Signaling Following Ischemia/Reperfusion in Skeletal Muscle of Old Mice

Abstract

With the advancement of age, skeletal muscle undergoes a progressive decline in mass, function, and regenerative capacity. Previously, our laboratory has reported an age-reduction in recovery and local induction of IGF-I gene expression with age following tourniquet (TK)-induced skeletal muscle ischemia/reperfusion (I/R). In this study, young (6 mo) and old (24 28 mo) mice were subjected to 2 hours of TK-induced ischemia of the hindlimb followed by 1, 3, 5, or 7 days of reperfusion. Real time-PCR analysis revealed clear age-related reductions and temporal alterations in the expression of IGF-I and individual IGF-I Ea and Eb splice variants. ELISA verified a reduction of IGF-I peptide with age following 7 days recovery from TK. Western blotting showed that the phosphorylation of Akt, mTOR, and FoxO3, all indicators of anabolic activity, were reduced in the muscles of old mice. These data indicate an age-related impairment of IGF-I expression and intracellular signaling does exist following injury, and potentially has a role in the impaired recovery of skeletal muscle with age.

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Document Details

Document Type
Technical Report
Publication Date
Apr 01, 2011
Accession Number
ADA619194

Entities

People

  • Christian Sell
  • David W. Hammers
  • J. S. Estep
  • Martin L. Adamo
  • Roger P. Farrar
  • Ronald W. Matheny Jr.
  • Thomas J. Walters

Organizations

  • United States Army Institute of Surgical Research

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Alkanes
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Medical Personnel
  • Muscle Fibers
  • Muscles
  • Myoblasts
  • Proteins
  • Ribonucleic Acids
  • Skeletal Muscle
  • Statistical Analysis
  • Stem Cells
  • Tissues
  • Transcription Factors

Fields of Study

  • Biology

Readers

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