Determining the Molecular and Genetic Basis for Diabetes in Navy Bottlenose Dolphins (Tursiops truncatus)

Abstract

CREB-ZF functions as a transcriptional represser, at least in terms of its effects on gluconeogenic genes. Efforts are underway to determine whether CREB-ZF directly interferes with activation of the cAMP-responsive factor CREB or the CRTC family of CREB coactivators. CREB-ZF has been reported to modulate the unfolded protein response (UPR) although the underlying mechanism is unclear. Because it contains a leucine zipper DNA binding domain, CREB-ZF would be expected to compete for binding to CREB binding sites. We will test whether CREB-ZF displaces CREB and CRTC2 from gluconeogenic promoters and thereby reduces hepatic glucose production.

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Document Details

Document Type
Technical Report
Publication Date
Jan 12, 2015
Accession Number
ADA620249

Entities

People

  • Marc R. Montminy

Organizations

  • Salk Institute for Biological Studies

Tags

Communities of Interest

  • Human Systems

DTIC Thesaurus Topics

  • Carrier Proteins
  • Diabetes
  • Families (Human)
  • Gene Expression
  • Glucose Metabolism Disorders
  • Insulin
  • Liver Diseases
  • Medical Personnel
  • Metabolic Diseases
  • Metabolism
  • Military Personnel
  • Polymerase Chain Reaction
  • Production
  • Proteins
  • Technology Transfer
  • Transcription Factors
  • Type 2 Diabetes

Fields of Study

  • Biology

Readers

  • Allergy and Immunology.
  • Breast cancer cell signaling and growth regulation.
  • Marine Mammal Biology

Technology Areas

  • Biotechnology