Biochemical Characterisation of TSC1 and TSC2 Variants Identifiedd in Patients with Tuberous sclerosis Complex

Abstract

The key findings of the project during the research period are as follows: 1. Derivation of 62 unclassified TSC2 variants: 27 classified as pathogenic; 10 classified as neutral; 27 still unclassified/analysis not complete. 2. Derivation of 20 unclassified TSC1 variants: 8 classified as pathogenic; 7 classified as neutral; 5 still unclassified/analysis not complete 3. Demonstration that TSC1 missense mutations cause TSC. 4. Identification of a region of TSC1 (amino acids 50 - 224) required for maintaining TSC1 at sufficient levels in the cell to form a stable TSC1-TSC2 complex and inhibit mTOR. 5. Identification of amino acid residues involved in (i) TSC1-TSC2 binding, and (ii) rhebGAP activity. 6. Robust assay for detection of pathogenic TSC2 variants. 7. Improvements in assay cost, throughput and reproducibility.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2008
Accession Number
ADA622174

Entities

People

  • Dicky Halley
  • Marianne Hoogeveen-westerveld
  • Mark Nellist

Organizations

  • Erasmus MC

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  • Amino Acids
  • Biomedical Research
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  • Genetic Code
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