Chemical Blistering: Cellular and Macromolecular Components
Abstract
The overall purpose of this investigation is to elucidate the molecular mechanisms by which bis(beta-chloroethyl) sulfide (BCES) exerts its vesicant action when applied topically to human skin. The technical objectives of the project are to establish morphological, cytochemical, and/or biochemical indicators of epidermal mustard toxicity and to examine and evaluate the relevance of these indicators to vesication. The following three biological systems have been used in this study: (a) stratified, cornified primary cultures of keratinocytes grown on nylon microporous membranes positioned at the air- medium interface; (b) submerged monolayer cultures of primary keratinocytes that contain early differentiated as well as germinating cells and are grown in a medium having a low level (0.08-0.10 micronmo) of Ca2(+)(Calcium ions); and (c) cultures of the BSC 40 line of monkey kidney cells grown as a submerged monolayer. Keywords: Mustard, Keratinocyte, Tissue culture, Alkylation, Toxicity, Chemical blistering, Metabolism, DNA Repair, Mitochondria, Epidermal differentiation, RAV, BCES, BSC.
Document Details
- Document Type
- Technical Report
- Publication Date
- Dec 31, 1989
- Accession Number
- ADB145621
Entities
People
- I. A. Bernstein
Organizations
- University of Michigan