Heterogeneity of Enzyme-Altered Foci in Rat Liver,
Abstract
Enzyme-altered foci (EAF) in liver are assumed to be precursor lesions for tumors in this organ. Results obtained with selected hepatocarcinogens which produce lesions of differing phenotype and growth behavior indicate that not the total number of enzyme-altered cells, but rather, the proliferation of individual cell clones is of major importance for additional changes leading to malignancy. Analyses including multiple marker enzymes demonstrate a relationship between foci phenotype and proliferation. The inducibility of certain down-regulated enzymes in EAF indirectly suggests disturbances in the expression of regulatory genes such as protooncogenes. Data on Ha-ras and c-myc protooncogene expression in EAF are presented.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 1990
- Accession Number
- ADP006365
Entities
People
- Albrecht Buchmann
- Dale Pearson
- Martina Schulte
- Michael Schwarz
- Werner Kunz
Organizations
- German Cancer Research Center