Methylation Mediated Repression of Selected Genes in Prostate Cancer

Abstract

The present project is focused on understanding the exact mechanism by which methylation silences gene expression in prostate cancer cell lines. Certain proteins bind preferentially to methylated DNA and these proteins have been shown to repress gene expression. In order to determine which of these protein or proteins interact with methylated genes inside the cells, we plan to use using chromatin immunoprecipitation assay. Two important requirements for this assay include an optimal sonication of the fixed chromatin (most of the sonicated fragments should be around 500bp) and quantitative PCR assay for the gene under study. We have determined the linear PCR conditions for the proposed genes as well as the optimal fixation and sonication conditions for the prostate cancer cell lines. We have performed chromatin immunoprecipitation assay for the GSTP1 and AR promoters in LNCaP prostate cancer cell lines. As compared to the AR promoter, the GSTP1 promoter was enriched in deacetylated histones H3 and H4. These results are consistent with the recruitment of histone deacetylase containing complexes by methylated DNA, resulting in a localized deacetylation.

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Document Details

Document Type
Technical Report
Publication Date
Apr 01, 2003
Accession Number
ADA416713

Entities

People

  • Rakesh Singal

Organizations

  • United States Department of Veterans Affairs

Tags

DTIC Thesaurus Topics

  • Abstracts
  • Androgen Receptors
  • Antibodies
  • Biomedical Research
  • Cancer
  • Carrier Proteins
  • Cell Line
  • Chemical Synthesis
  • Chemistry
  • Department Of Veterans Affairs
  • Methylation
  • Neoplasms
  • Prostate
  • Prostate Cancer
  • Proteins
  • Tissue Culture
  • Tissues

Fields of Study

  • Biology

Readers

  • Molecular and Cellular Biochemistry
  • Molecular and genetic basis of cancer.
  • Prostate Cancer Biology.